Salk Research: Spike Proteins Alone Cause Damage

Salk researchers released a paper on April 30 titled “SARS-CoV-2 Spike Protein Impairs Endothelial Function via Downregulation of ACE 2”. There is also a corresponding article to the paper.


Basically, the researchers exposed hamsters to a “noninfectious pseudovirus” that has the spike proteins which are characteristic of SARS-CoV-2. These spike proteins themselves—not the full virus—caused damage to the lungs.


It has been mentioned by doctors during the trials of the so-called vaccine that the generated antibodies which are meant to act against the coronavirus spike protein may cause further problems because they are non-neutralizing antibodies. This is not news.


Nevertheless, this latest research from Salk combined with the serious doubts regarding the so-called vaccine makes it even more worrying. If the so-called vaccine (which isn’t truly a vaccine) tells one’s body to make the spike protein in order to trigger the body to make the antibodies against the spike proteins… well, with all the spike proteins floating around (regardless of their origin), what do they do to one’s body? And if the antibodies don’t do their job properly because they are non-neutralizing antibodies, then what effect will all the spike proteins continue to have?


SARS-CoV-2 Spike Protein Impairs Endothelial Function via Downregulation of ACE 2
Figure A: Representative H&E histopathology of lung specimens from 8- to 12 wk-old male Syrian hamsters 5-day post administration of pseudovirus overexpressing Spike protein (Pseu-Spike) or mock virus in control group (n=3 mice per group, 1×108 PFU). Thickened alveolar septa (red arrowhead) and mononuclear cell (red arrow). Scale bar=20 μm.
 

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